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daniela


ana gabriela henriques
biographical description

Graduated in Biology at the University of Aveiro and did a MSc and PhD on the molecular basis of Alzheimer’s disease. She is invited assistant professor at the Health Sciences Department of the University of Aveiro, where she teaches biomarkers in biomedicine and clinical pathology. She is vice-director of the Master’s degree in Molecular Biomedicine.

e-mail: aghenriques@ua.pt  | tel: +351 234 370 200 506

bibliometry
http://orcid.org/0000-0003-0851-6979

research interests
I am interested in disease biomarker discovery. My goal is to identify new biomarkers for the clinical diagnostics of dementia. Dementia diagnosis is difficult, in particular during the early stages or when symptoms overlap across dementia subtypes. Alzheimer´s disease (AD) is the most common form of dementia with high incidence in the elderly population. I have a particular interest in AD differential diagnosis and prognostics. For this, I am organizing a longitudinal cohort in the Aveiro region (the Av-cohort), which is essential for clinical biomarkerdiscovery. I am particularly interested on neuro inflammation and phosphorylation associated biomarkers.

selected publications
1.
Henriques A.G., Müller T., Oliveira J.M., Cova M., da Cruz e Silva C.B., da Cruz e Silva O.A. (2016). Altered protein phosphorylation as a resource for potential AD biomarkers. Scientific Reports. doi: 10.1038/srep30319

 2. Henriques A.G., Oliveira J.M., Carvalho L. and da Cruz e Silva O.A.B. (2015). Abeta influences cytoskeletal signalling cascades with consequences to Alzheimer´s disease. Molecular Neurobiology, 52,1391-407. doi: 10.1007/s12035-014-8913-4.

3. Henriques A.G., Oliveira J.M., Gomes B., Ruivo R., da Cruz e Silva E.F. and da Cruz e Silva O.A.B. (2014). Complexing Abeta prevents the cellular anomalies induced by the peptide alone. Journal of Molecular Neuroscience 53, 661-668. doi: 10.1007/s12031-014-0233-7

4. Henriques A.G., Vieira S. I., da Cruz e Silva E. F. and da Cruz e Silva O.A.B. (2010). Aβ promotes Alzheimer’s disease-like cytoskeleton abnormalities with consequences to APP processing in neurons. J Neurochem. 113, 761-771. doi: 10.1111/j.1471-4159.2010.06643.x

5. Henriques A. G., Vieira S.I., da Cruz e Silva E. F. and da Cruz e Silva O.A.B. (2009). Abeta hinders nuclear targeting of AICD and Fe65 in primary neuronal cultures. J. Mol. Neurosci. 39, 248-55. doi: 10.1007/s12031-009-9192-9

última atualização a 28-09-2016
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